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1) FURTHER REFINEMENT: Thank you for bearing with me. This is difficult.
HUMAN ACETYLCHOLINE DEFICIENCY DISEASE
SARS-CoV-2 induces a progressive (fatal?) autoimmune disease which destroys Nicotinic Acetylcholine Receptors. The disease is induced when the spike protein toxifies
HUMAN ACETYLCHOLINE DEFICIENCY DISEASE
SARS-CoV-2 induces a progressive (fatal?) autoimmune disease which destroys Nicotinic Acetylcholine Receptors. The disease is induced when the spike protein toxifies
2) the Nicotinic Acetylcholine Receptors by hyperactivating them. This causes the immune system to relentlessly destroy nAChRs. The main symptom of the disease is a Acetylcholine Deficiency, as its uptake is inhibited by the destruction of nAChRs.
3) The Symptoms of Acetylcholine Deficiency (Long COVID):
Memory problems
Fast heart rate
Difficulty with word recall when speaking
Learning difficulties
Dry mouth
Dry eyes
Orthostatic hypotension
Low muscle tone
Depression
Chronic inflammation
Emotional instability
Constipation
Memory problems
Fast heart rate
Difficulty with word recall when speaking
Learning difficulties
Dry mouth
Dry eyes
Orthostatic hypotension
Low muscle tone
Depression
Chronic inflammation
Emotional instability
Constipation
4) This explains why we are seeing an increase in diabetes post-COVID. The receptors are being destroyed and the increased free Acetylcholine is inducing diabetes. This is what happens to β2-nAChR-knockout mice. https://academic.oup.com/endo/article/155/10/3793/2423081
5) Epithelial cells, immune cells, mesothelial cells, and mesenchymal cells all produce Ach. Since the body believes it needs more Ach, these cells begin to release Ach. This results in massive vasodilation. The body responds with a massive coagulation response to stop "leaking."
6) https://pubmed.ncbi.nlm.nih.gov/27730645/
Inexorably, when enough nAChRs are destroyed, the parasympathetic nervous system can no longer counterbalance the sympathetic nervous system. At this point any startling event, such as a dream or adrenaline rush, will elicit a fatal arrhythmia.
Inexorably, when enough nAChRs are destroyed, the parasympathetic nervous system can no longer counterbalance the sympathetic nervous system. At this point any startling event, such as a dream or adrenaline rush, will elicit a fatal arrhythmia.
7) Evidence this is the cause of MIS-C and MIS-A. I believe we will see a massive incidence of these post vaccination.
...paradoxical vasoconstriction in response to acetylcholine https://www.ahajournals.org/doi/full/10.1161/cir.0000000000000484
...paradoxical vasoconstriction in response to acetylcholine https://www.ahajournals.org/doi/full/10.1161/cir.0000000000000484
8) There is evidence that what killed the doctor in Florida -Immune Thrombocytopenia Purpura and Kawasaki Disease are causally related by the presence of ACHR AUTOANTIBODIES! https://www.frontiersin.org/articles/10.3389/fimmu.2020.00496/full
9) Indeed, nAChR knockout mice have an irreversible antagonist effect. Resulting in precisely what I have hypothesized.
Specific genes encoding for receptors are
deleted, thus, in essence, providing a highly selective, albeit irreversible, "antagonist." https://link.springer.com/chapter/10.1007/978-3-642-57079-7_20
Specific genes encoding for receptors are
deleted, thus, in essence, providing a highly selective, albeit irreversible, "antagonist." https://link.springer.com/chapter/10.1007/978-3-642-57079-7_20
