1) This is why scientific debate is SO IMPORTANT. With censorship of scientific and medical professionals, innovation and discovery is STUNTED! Maybe they want that? In a discussion with @flavinkins something became clear immediately. As I have mentioned throughout...
2) ADE - antibody-dependent-enhancement...And when a subject exhibits this phenomenon post vaccine, will they will call it COVID-21 and make a new bogus PCR, and say it’s yet another virus causing this (not the vaccine)???Hmmm could be- https://www.biorxiv.org/content/10.1101/2020.12.18.423358v1?fbclid=IwAR3nCWCjmMMxO8Ub9E0-IDDJxJa0Fvbs2PxXxTkSdC-evI5k4maQmJptVxo
3) This was what happened with the SARS(1) “vaccine” animal models (ADE). But they jumped straight to humans this go-round. A scientific “don’t ask, don’t tell”.
Need to analyze the severe Covid patients (apparently all hospitalized had the ADE antibodies) https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3335060/
Need to analyze the severe Covid patients (apparently all hospitalized had the ADE antibodies) https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3335060/
4) this could be the hallmark of the “high risk”... interrupt this N-terminus AB, decrease severity of infection... You want an effective, “vaccine” mitigating ADE? Bind these antibodies and decrease binding affinity to the levels of the mild and asymptomatic cases....
5) highly likely one of the modalities as to why Hydroxychloroquine showed benefit, particularly early/pre-exposure. It appears as though N-terminus Antibody concentration is proportional to disease severity. @zev_Zelenko @JamesTodaroMD @drsimonegold @dockaurG @ScottWAtlas