Read on Twitter
Daily reminder:
— Coronavirus main host is a bat
— Bats are not found in Wuhan/Hubei region
— Bats would have been hibernating around time of the outbreak
— COVID19 is extremely susceptible to both heat and UV light
— Bioinformatics data storing genetic
sequence of a
— Coronavirus main host is a bat
— Bats are not found in Wuhan/Hubei region
— Bats would have been hibernating around time of the outbreak
— COVID19 is extremely susceptible to both heat and UV light
— Bioinformatics data storing genetic
sequence of a
coronavirus back in 2013, had its name changed by the Chinese — a deliberate attempt to coverup the fact that the virus shares a large percentage of the sequence of COVID19
of COVID19
— COVID19 can only spread indoors. This is not to say it cannot eventually evolve to spread outdoors but nonetheless evolved to spread indoors. This is highly conspicuous and indicative of laboratory origin
— Genetic sequence of COVID19 indicates that the virus is a chimera
— Genetic sequence of COVID19 indicates that the virus is a chimera
Meaning its genetic sequence is made up of more than one type of virus. This is evident when analyzing the spike protein. The spike protein is an integral protein that allows the virus to attach to the ACE2 receptor in humans.
S Protein = Spike Protein
S Protein = Spike Protein
When analyzing the sequence of the spike protein of COVID19 it becomes apparent the virus is a chimera of two viruses:
— Spike protein of ancestral bat strain RaTG13
— However the receptor binding motif (RBM) in its spike protein is replaced by the RBM from a pangolin strain
— Spike protein of ancestral bat strain RaTG13
— However the receptor binding motif (RBM) in its spike protein is replaced by the RBM from a pangolin strain
In addition to the swap of RBM:
— 4 amino acids are inserted. This creates a furin cleavage site.
What does all of this do? As previously explained by virologists—significantly expands the “repertoire” of the virus and allows it to infect more types of cells
— 4 amino acids are inserted. This creates a furin cleavage site.
What does all of this do? As previously explained by virologists—significantly expands the “repertoire” of the virus and allows it to infect more types of cells
Why is this important?
— The furin site allowed the virus to “jump” from bats to humans
— This ability to jump from bats to humans along with the near impossibility of the genetic change occurring in humans indicates lab manipulation
— For the virus to occur naturally it would
— The furin site allowed the virus to “jump” from bats to humans
— This ability to jump from bats to humans along with the near impossibility of the genetic change occurring in humans indicates lab manipulation
— For the virus to occur naturally it would
need to have an intermediate animal host, where it evolved and then gained the capability to infect humans. There is no indication or evidence of an intermediate animal host.
This came from a lab. This came from the Wuhan lab.
This came from a lab. This came from the Wuhan lab.
The Wuhan lab had been working on chimera viruses since 2007. There is no doubt or question on whether or not they had the capability of doing this. They have been doing this for 13 years. The question is, why cover it up and who else was associated with the lab?!
The head of coronavirus research at the Wuhan Institute of Virology, Shi Zhengli, has done plenty of work replacing RBM in one virus and substituting it with the RBM from another virus. Some of the work also includes adding a furin site to these chimera viruses.
From 2007-2017, a total of at least 8 chimeric coronaviruses were developed at the Wuhan Lab. As recently as 2019, the Wuhan Lab received a 3.7 million dollar grant from the NIH. President Trump put a stop to this and it was not his administration who started this.
A paper from Shi Zhengli in 2019 makes some dubious claims regarding the justification of this grant: https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7097006/
https://threadreaderapp.com/thread/1291244581858803713.html
Twitter conveniently trimmed half this damn thread in half
but this link still has the full details
Twitter conveniently trimmed half this damn thread in half
but this link still has the full details
Just got clarification’s sake, the flu did not “disappear.” People have been incorrectly filtering CDC’s data by excluding Jan-April 2020 and only looking at Oct-Dec 2020. Also keep in mind flu does not to be lab confirmed to be officially counted.
For*
Flu season runs from Oct-April so there is overlap over two different years.
So for 2020 you would need:
October 2019 - December 2019
January 2020 - April 2020
This wouldn’t be readily apparent to most. It should to doctors pushing the “flu disappeared” nonsense
So for 2020 you would need:
October 2019 - December 2019
January 2020 - April 2020
This wouldn’t be readily apparent to most. It should to doctors pushing the “flu disappeared” nonsense
Propaganda used by MSM:
— Social Distancing (that was called good manners back in February)
— Asymptomatic Spread: Absolutely impossible. Many symptoms are manifestations of the immune response to a pathogen, not just the pathogen itself.
How would asymptomatic spread be
— Social Distancing (that was called good manners back in February)
— Asymptomatic Spread: Absolutely impossible. Many symptoms are manifestations of the immune response to a pathogen, not just the pathogen itself.
How would asymptomatic spread be
remotely possible? It’s like someone telling you the sky is purple. Ridiculous lie
“Flatten the curve” then show you a generic viral life cycle graph and made up an unattainable standard
“Flatten the curve” then show you a generic viral life cycle graph and made up an unattainable standard
RaTG13 strain (green) shares a high degree of homology with COVID19 (red). Where the peaks spike is where homology is shared.
Where these two strains differ is the RBM. This is because an RBM from pangolin strain was combined with spike protein from RaTG13
Where these two strains differ is the RBM. This is because an RBM from pangolin strain was combined with spike protein from RaTG13
