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Prof Francois Balloux
BallouxFrancois
We previously analysed recurrent #SARSCoV2 mutations for an association with increased transmission using ~47k genomes. At the time, we found no evidence for any mutation making the virus intrinsically more
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Some random thoughts on caused for the rise of the #SARSCoV2 'UK lineage' (B 1.1.7). If its increase in frequency in the UK were caused by intrinsically higher contagiousness. It
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I felt it may be helpful to provide an update on the new #COVID19 lineages in the UK and in SA that might both be more contagious than any #SARSCoV2
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I'm a bit puzzled that some still deny the concept of 'herd immunity' despite the incoming vaccines. Please let me state this one more time, there's nothing political about herd
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New preprint out. We investigated whether we could detect evidence for genetic recombination in #SARSCoV2, and found none to date. We used (slightly tweaked) standard statistical genetics methods, extensive simulations
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"#SarsCov2 has never been isolated" strikes me as arguably the most absurd of all the '#COVID19 myths', and this in the face of stiff competition on all sides of the
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To give some context to the recent announcement of a new #SARSCoV2 variant/mutation, it may be helpful to realise that in the paper below we identified >12,000 variants/mutations in the
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I believe the incoming #COVID19 vaccines tend to be effective and safe. I also predict widespread vaccination campaigns will save lives. Though, I doubt vaccines will significantly shorten the timeline
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New preprint. We assessed the source of T-cell X-reactivity to #SARSCoV2 in unexposed people. Previous exposure to 'common cold' endemic HCoVs plays at most a marginal role, and we found
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Vaccine safety and persistent post-infection symptoms (long-COVID) are both important issues that require our scrutiny and attention. Though, inventing and spreading lurid and fanciful #COVID19 scare stories (e.g. exploding hearts
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Antibody-Dependent Enhancement (ADE) is a complex and poorly understood phenomenon, likely underpinned by different mechanisms, where binding of a virus to suboptimal antibodies enhances severity of the disease, rather than
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Paper out in Nature Comms. We developed a framework to analyse the contribution of #SARSCoV2 mutations to the virus' transmissibility. We applied it to nearly 50k genomes and we found
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